statistiche ricercatore

Continente	#
NA - Nord America	1.192
AS - Asia	677
EU - Europa	429
SA - Sud America	63
AF - Africa	13
Continente sconosciuto - Info sul continente non disponibili	1
Totale	2.375

Nazione	#
US - Stati Uniti d'America	1.161
SG - Singapore	247
CN - Cina	178
IT - Italia	92
DE - Germania	77
VN - Vietnam	74
GB - Regno Unito	65
BR - Brasile	52
PL - Polonia	51
HK - Hong Kong	44
FI - Finlandia	33
TR - Turchia	33
UA - Ucraina	31
IN - India	24
CA - Canada	21
FR - Francia	19
ID - Indonesia	18
SE - Svezia	16
JP - Giappone	15
BE - Belgio	12
RU - Federazione Russa	11
BD - Bangladesh	8
MX - Messico	8
AR - Argentina	5
MY - Malesia	5
NL - Olanda	5
IQ - Iraq	4
PK - Pakistan	4
UZ - Uzbekistan	4
ZA - Sudafrica	4
AT - Austria	3
CZ - Repubblica Ceca	3
EC - Ecuador	3
JO - Giordania	3
KE - Kenya	3
KR - Corea	3
SA - Arabia Saudita	3
AL - Albania	2
AZ - Azerbaigian	2
DZ - Algeria	2
ES - Italia	2
ET - Etiopia	2
KG - Kirghizistan	2
RO - Romania	2
AF - Afghanistan, Repubblica islamica di	1
CH - Svizzera	1
CO - Colombia	1
CR - Costa Rica	1
GF - Guiana Francese	1
HR - Croazia	1
IL - Israele	1
IR - Iran	1
JM - Giamaica	1
KZ - Kazakistan	1
LR - Liberia	1
LT - Lituania	1
NG - Nigeria	1
PE - Perù	1
PH - Filippine	1
PT - Portogallo	1
RS - Serbia	1
TH - Thailandia	1
XK - ???statistics.table.value.countryCode.XK???	1
Totale	2.375

Nome	#
Regulation of a strong F9 cryptic 5'ss by intrinsic elements and by combination of tailored U1snRNAs with antisense oligonucleotides	299
An exon-specific U1snRNA induces a robust factor IX activity in mice expressing multiple human FIX splicing mutants	295
Exploring Splicing-Switching Molecules For Seckel Syndrome Therapy	280
Genetic determinants of activated factor VII antithrombin complex plasma concentration include tissue factor, factor VII and endothelial protein C receptor gene variants	246
A unique exon specific U1snRNA rescues different haemophilia B - causing splicing-defective factor IX variants in mice	217
Correction of aberrant splicing causing haemophilia B through the combination of compensatory U1snRNAs and antisense oligonucleotides	198
Disease-causing variants of the conserved+2T of 5 ' splice sites can be rescued by engineered U1snRNAs	181
The somatic FAH C.1061C>A change counteracts the frequent FAH c.1062+5G>A mutation and permits U1snRNA-based splicing correction	178
A Compensatory U1snRNA Partially Rescues FAH Splicing and Protein Expression in a Splicing-Defective Mouse Model of Tyrosinemia Type I	177
Rescue of missense and splicing mutations in Haemophilia A by a unique Exon Specific U1snRNA	175
Regulation of exon definition by intrinsic elements and by combination of tailored U1snRNA with antisense oligonucleotides	173
Totale	2.419

	Totale	Lug	Ago	Sett	Ott	Nov	Dic	Gen	Feb	Mar	Apr	Mag	Giu
2020/2021	12	0	0	0	0	0	0	0	0	0	0	0	12
2021/2022	138	8	18	1	6	5	8	9	3	6	12	8	54
2022/2023	146	15	10	10	17	21	14	13	15	14	1	13	3
2023/2024	77	2	9	0	1	5	13	4	15	1	1	1	25
2024/2025	366	6	7	35	20	41	39	12	7	76	36	48	39
2025/2026	896	85	24	75	95	128	62	156	57	75	97	29	13
Totale													2.419

SFERA Archivio dei prodotti della Ricerca dell'Università di Ferrara