SFERA Archivio dei prodotti della Ricerca dell'Università di Ferrara

TESTA, Maria Francesca
 Distribuzione geografica
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Continente #
NA - Nord America 455
AS - Asia 149
EU - Europa 142
AF - Africa 2
SA - Sud America 1
Totale 749
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Nazione #
US - Stati Uniti d'America 449
SG - Singapore 63
IT - Italia 61
CN - Cina 43
DE - Germania 20
FI - Finlandia 20
ID - Indonesia 18
CZ - Repubblica Ceca 9
IQ - Iraq 7
TR - Turchia 7
CA - Canada 6
SE - Svezia 6
HK - Hong Kong 5
NL - Olanda 5
FR - Francia 4
GB - Regno Unito 4
RU - Federazione Russa 4
UA - Ucraina 4
IN - India 2
NG - Nigeria 2
TW - Taiwan 2
BE - Belgio 1
BR - Brasile 1
CH - Svizzera 1
EE - Estonia 1
GE - Georgia 1
JP - Giappone 1
LT - Lituania 1
NO - Norvegia 1
Totale 749
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Città #
Santa Clara 68
Singapore 56
Fairfield 50
Chandler 48
Ferrara 28
New York 26
Ashburn 24
Seattle 22
Helsinki 18
Jakarta 18
Wilmington 18
Houston 16
Cambridge 15
Woodbridge 14
Shanghai 12
Princeton 10
Los Angeles 9
Brno 8
Milan 8
San Diego 8
Baghdad 7
Bremen 7
Falkenstein 6
Izmir 6
Dearborn 5
Naples 5
Redwood City 5
Bologna 4
Florence 4
Jacksonville 4
Rochester 4
Rome 4
San Mateo 4
Hong Kong 3
Kunming 3
London 3
Munich 3
Nanjing 3
Philadelphia 3
Ann Arbor 2
Beijing 2
Guangzhou 2
Henderson 2
Jiaxing 2
Lagos 2
Lappeenranta 2
Monmouth Junction 2
Moscow 2
Noventa Vicentina 2
Ottawa 2
Padova 2
Shenyang 2
Taipei 2
The Hague 2
Thornhill 2
Toronto 2
Worcester 2
Ambrolauri 1
Andover 1
Boardman 1
Brussels 1
Central District 1
Changsha 1
Dallas 1
Ferrara di Monte Baldo 1
Hefei 1
Jinan 1
Kanpur 1
Kowloon City 1
Leawood 1
Mountain View 1
Mumbai 1
Mégevette 1
Nanchang 1
Norwalk 1
Olomouc 1
Orange 1
Oslo 1
Piemonte 1
Sayreville 1
Solihull 1
Southgate 1
São Paulo 1
Tallinn 1
Tokyo 1
Washington 1
Totale 624
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Nome #
Secretion of wild-type factor IX upon readthrough over F9 pre-peptide nonsense mutations causing hemophilia B 110
Secretion of wild-type factor IX upon readthrough over F9 pre-peptide nonsense mutations causing hemophilia B 99
TRPP2 dysfunction decreases ATP-evoked calcium, induces cell aggregation and stimulates proliferation in T lymphocytes 88
Exploring spontaneous readthrough over recurrent F8 nonsense mutations: potential correlation with inhibitor risk? 57
Molecular insights into determinants of translational readthrough and implications for nonsense suppression approaches 54
Translation termination codons in protein synthesis and disease 52
Detection of Residual Factor VIII Levels Reveals the Occurrence of Readthrough Over the Majority of F8 Nonsense Mutations 49
An advanced method for the small-scale production of high-quality minicircle DNA 44
Spontaneous readthrough over recurrent F8 nonsense mutations is associated with residual factor VIII levels: implications for inhibitor risk? 41
Comparative Analysis Of Residual Factor VIII Expression from Recurrent F8 Nonsense Mutations Indicates that Localization in the B- domain Favours Readthrough- mediated Protein Output 35
Translational readthrough at F8 nonsense variants in the factor VIII B domain contributes to residual expression and lowers inhibitor association 35
An engineered factor X variant as a novel by-passing agent for hemophilia 30
A factor X variant with engineered activation peptide as innovative by-passing agent for haemophilia 30
The p.P1127S pathogenic variant lowers von Willebrand factor levels through higher affinity for the macrophagic scavenger receptor LRP1: Clinical phenotype and pathogenic mechanisms 26
Engineered suppressor tRNAs as a novel correction approach for recurrent hemophilia A-causing nonsense mutations 16
An integrated multi-tool analysis contributes elements to interpreting unclassified factor IX missense variants associated with haemophilia B 15
Tailored collagen binding confers distinct functional properties to engineered factor IX fusion proteins in hemophilia B mouse model 7
Design of an engineered coagulation factor X as a novel by-passing agent for hemophilia 6
The heterozygous p.R854Q variant is responsible for the conformational changes in the D’D3 domain of the von Willebrand Factor, by weakening its interaction with factor VIII: in-silico analysis 4
A novel tailored correction approach for recurrent hemophilia-causing nonsense mutations through AntiCodon-Engineered suppressor tRNAs 3
Engineered tRNAs as a novel tailored correction approach to restore protein biosynthesis and function from hemophilia-causing nonsense mutations 3
Reverting highly frequent f8 nonsense mutations: base and prime editing approach for hemophilia A 2
Base and Prime Editors as new correction approaches for recurrent Hemophilia A Nonsense Mutations 2
Von Willebrand factor natural variants of the arginine 1205: in-silico analysis of the protein structural changes and its interactions with the macrophagic scavenger receptor LRP1 1
Totale 809
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Categoria #
all - tutte 5.969
article - articoli 0
book - libri 0
conference - conferenze 0
curatela - curatele 0
other - altro 0
patent - brevetti 0
selected - selezionate 0
volume - volumi 311
Totale 6.280
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Totale Lug Ago Sett Ott Nov Dic Gen Feb Mar Apr Mag Giu
2019/202045 0 0 0 0 0 0 16 12 9 4 1 3
2020/202170 4 4 1 3 1 5 18 14 3 4 12 1
2021/202280 17 7 1 6 1 3 1 2 4 7 3 28
2022/2023115 11 14 5 9 16 8 11 11 14 2 4 10
2023/2024128 6 6 7 1 4 33 4 10 10 4 5 38
2024/2025265 23 8 47 16 58 112 1 0 0 0 0 0
Totale 809